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Inactivation associated with AKT/NF‑κB signaling by eurycomalactone diminishes man NSCLC mobile or portable stability as well as increases the chemosensitivity to cisplatin.
Inactivation associated with AKT/NF‑κB signaling by eurycomalactone diminishes man NSCLC mobile or portable stability as well as increases the chemosensitivity to cisplatin.